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This includes NOX, apurinic/apyrimidinic endonuclease 1 (APE1), Prx, thioredoxin (TRX)-related transmembrane-2 (TMX2), activator protein 1 (AP-1), PDI, and SOD1

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[DOI] [PubMed] [Google Scholar] 36.Duthie S.J., Narayanan S., Brand G.M., Pirie L., Grant G

Further investigation revealed that the IC50 concentration of CGA, through the depletion of free iron within hepatocytes, significantly reduced the expression of iron-sulfur cluster subunits in mitochondrial complexes I-III and disrupted the oxidative-reductive homeostasis of hepatocytes, ultimately leading to hepatotoxicity