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By upregulating -AR and supporting HSL phosphorylation, AOD9604 is therefore thought to support a more robust lipolytic response in visceral adipocytes where those receptors and enzymes are most abundant

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Reduction of blood glucose levels and suppression of G6pc expression in liver of Insr P1195L/ +/HFD mice by supplementation with either CA or UDCA suggest that alterations in BAs might contribute to the development of hyperglycemia in these mice