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Additionally, we identify the FHA domain of SNIP1 as a reader of P-SF3B1, guiding the recruitment and assembly of the RES complex during spliceosome activation, and show that mutations in the FHA domain, including the neurodevelopmental disorder-linked E366G variant, impair this interaction, resulting in defects in splicing and cellular proliferation

Non-covalent inhibitors of TrxR class of redox proteins may have broad application One of our recently synthesized TGR inhibitors compound 10 with IC 50 = 18.6 M for TGR is even more potent against TrxR from Brugia malayi (BmTrxR